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Body fat not always bad

With the threat of an obesity crisis looming, a study led by UCL researchers reveals today that fat tissue isn’t always the enemy. Reporting in the journal Science they show that a molecular signalling pathway in fat tissue is an important mediator in extending lifespan.

11 Jun 2004

Higher-than-normal amounts of a selenium-containing enzyme could promote type 2 diabetes

A study by researchers at Cornell University suggests that higher-than-normal amounts of a selenium-containing enzyme could promote type 2 diabetes.

10 Jun 2004

High vitamin C intake may protect against rheumatoid arthritis

A high vitamin C intake may protect against the development of painful widespread rheumatoid arthritis indicates research published in the Annals of the Rheumatic Diseases.

10 Jun 2004

Genetic signature associated with the aging human brain

Unraveling the mysteries of the aging brain is a major goal for brain science, especially given the exploding population of senior citizens and the obvious desire to preserve brain function as long as possible.

9 Jun 2004

Mice may hold the key to longer life

Scientists from the University of Aberdeen, the Aberdeen-based Rowett Research Institute and the Medical Research Council (MRC) in Cambridge have made a major breakthrough in understanding how metabolism affects lifespan.

2 Jun 2004

Data suggests potential target for Alzheimer's disease treatment

Researchers from Weill Cornell Medical College and Columbia University College of Physicians & Surgeons have discovered at least one way in which beta-amyloid, the protein that forms stubborn plaques in the brains of Alzheimer's disease patients, can kill nerve cells.

25 May 2004

Protein that appears to play a key role in Alzheimer's disease

A protein that allows human cancer to resist multiple anticancer drugs also appears to play a key role in Alzheimer's disease, according to research conducted at Fox Chase Cancer Center. The protein, active in brain tissue, could be a target for new drugs to treat patients with Alzheimer's.

20 May 2004

Glucose metabolism could be used to selectively destroy cancer cells

Scientists have known for decades that cancer cells consume more glucose than normal cells. A longstanding assumption that the excess glucose metabolism was needed to make energy has not been borne out by research studies.

15 May 2004

The First International Congress on Antioxidant Methods

The First International Congress on Antioxidant Methods will be held June 16-18 in Orlando, Fla., in an effort to reach agreement on techniques for measuring and reporting on antioxidants, beneficial compounds found in foods, cosmetics and certain vitamins that are believed to fight cancer, heart disease and aging.

6 May 2004

Study suggests that the vitamins, such as E, C, and beta carotene, could raise bad form of cholesterol

The notion that antioxidant vitamins could provide a safe, convenient way to protect the heart from disease appears to have hit a pothole.

3 May 2004

Sprouts and broccoli may reduce risk of high blood pressure

Glucoraphanin, also known as sulforaphane glucosinolate (SGS(TM)), a naturally-occurring compound found in broccoli sprouts and broccoli, may reduce risk of high blood pressure, cardiovascular disease and stroke, according to new research published in the May 4 issue of the Proceedings of the National Academy of Sciences of the USA.

28 Apr 2004

Curry offers possible protection against Alzheimer’s disease

A new study has found that curry, a common and popular cooking additive, could be an effective enhancer of an enzyme that protects the brain against oxidative conditions.

19 Apr 2004

Possible new treatment strategy for Alzheimer's disease

A new study in mice identifies one of the missing steps in how Alzheimer's develops and suggests a possible new treatment strategy, according to researchers at Columbia University Medical Center and Weill Cornell Medical College and their colleagues.

16 Apr 2004

Certain prostate cancer cell lines are unable to repair DNA damage caused by "free radicals"

16 Apr 2004

DNA free radical damage research

Certain prostate cancer cell lines are unable to repair DNA damage caused by "free radicals," according to scientists at the National Institute of Standards and Technology (NIST) and National Institutes of Health (NIH). This type of damage has been implicated before in the development of prostate cancer, but the new research provides the first solid evidence that the normal repair process is altered in prostate cancer cells, possibly leading to a cascade of events that culminate in further DNA damage and cellular dysfunction.

9 Apr 2004