1. Phil Ip Phil Ip United States says:

    Hi Nate - thanks for your insights.  As a layman with only a few years of life sciences, I'm looking to ask someone a fundamental question: is it possible the mutation in P272L was merely from Proline unavailability and Leucine was "grabbed" during replication?  I know Leucine is an essential amino and Proline is synthesized from various glutamates, BUT if competition for Proline (perhaps due to an mRNA vaccine being given to a silently infected covid patient, as we know covid-19 impairs mitochondrial dyanmics) existed in even certain tissues/cells, could that not force the mutation?

    If you're able to answer, please take a shot.

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